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NUTRITIONAL APPROACHES TO SCHIZOPHRENIA
Vitamin E
It has been suggested that the chronic use of neuroleptic drugs may generate free radicals which damage synaptic terminals and destroy essential fatty acids. Vitamin E, which can restrict the formation of free radicals, produced an improvement in tardive dyskinesia.
It may be that the best results in schizophrenia, with or without tardive dyskinesia, may be achieved by combining essential fatty acids with Vitamin E. In the above study Vitamin E was added to both the Efamol and the placebo capsules.
Other nutritional approaches to schizophrenia: diet, zinc and B6
Cutting out wheat, milk and foods containing arachidonic acid (meat, dairy products and peanuts) has also been of help in some patients. According to the late Dr Carl Pfeiffer, most schizophrenics respond well to therapy with zinc and B6, in which they are often deficient. It could be that a combination of all these regimes is the most effective treatment. Drugs are effective, particularly on the ‘positive’ aspects of schizophrenia, such as hallucinations and bizarre behaviour, but they do little for the ‘negative’ symptoms, the withdrawal and the lack of emotional contact. The nutritional approaches, in contrast, seem to do little for the ‘positive’ symptoms but help the ‘negative’ ones. There is therefore a case for the two approaches together.
Caution: In contrast to schizophrenia, temporal lobe epilepsy may be made much worse by supplementation with evening primrose oil. Temporal lobe epilepsy and schizophrenia may be very difficult to distinguish from each other. However, the worsening of temporal lobe epilepsy in response to taking supplements of fatty acids means that giving EFA supplements could be the basis of a new diagnostic test. It is possible that many patients diagnosed as having schizophrenia in fact have temporal lobe epilepsy. If such patients are put on the appropriate drug, carbamazepine, they show a dramatic improvement which may mean they can leave hospital.
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